Age related cancer risk increases significantly with advancing age due to cumulative biological changes. Over time, cells accumulate genetic mutations, epigenetic alterations, and chromosomal instability. Declining DNA repair efficiency allows damaged cells to persist. At the same time, immune surveillance weakens, reducing the ability to detect and eliminate abnormal cells. Chronic inflammation and senescent cell accumulation further create a tissue environment that favors malignant transformation. Age related cancer risk reflects interactions between aging biology and environmental exposures rather than single causative factors. Understanding how aging increases cancer susceptibility supports prevention strategies focused on maintaining genome integrity, immune competence, and tissue homeostasis rather than treating cancer as an isolated disease.
Title : Change your genes – Change your life: Epigenetics of longevity
Kenneth R Pelletier, University of California School of Medicine, United States
Title : Improving mobility and health in over 45,000 humans using nanomedicine
Thomas J Webster, Brown University, United States
Title : An introduction to alchemical facial acupuncture: Sparking the shen
Mary Elizabeth Wakefield, Chi-Akra Center for Ageless Aging, United States
Title : Decoding the secret of longevity through big data: Noncoding RNAs—not proteins—drive animal lifespan evolution
Anyou Wang, DIFIBER LLC, United States
Title : Aspirin guided by coronary artery calcium scoring for primary prevention in persons with subclinical coronary heart disease
Arthur J Siegel, McLean Hospital, United States
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Narendra Kumar, HeartbeatsZ Academy, United Kingdom